Difference between revisions of "BsrA"

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(References)
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|style="background:#ABCDEF;" align="center"|'''Function''' || antagonist of [[SigA]]
 
|style="background:#ABCDEF;" align="center"|'''Function''' || antagonist of [[SigA]]
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|colspan="2" style="background:#FAF8CC;" align="center"| '''Interactions involving this RNA in [http://cellpublisher.gobics.de/subtinteract/startpage/start/ ''Subt''Interact]''': [http://cellpublisher.gobics.de/subtinteract/interactionList/2/BsrA BsrA]
 
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|style="background:#ABCDEF;" align="center"| '''Gene length''' ||  190 bp
 
|style="background:#ABCDEF;" align="center"| '''Gene length''' ||  190 bp
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=References=
 
=References=
'''Additional publications:''' {{PubMed|21881410,22102588}}
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'''Additional publications:''' {{PubMed|22333917,21881410,22102588}}
 
<pubmed>12177353 15811922</pubmed>
 
<pubmed>12177353 15811922</pubmed>
  
 
[[Category:small RNA]]
 
[[Category:small RNA]]

Revision as of 18:29, 20 February 2012

  • Description: 6S RNA

Gene name bsrA
Synonyms
Essential no
Product 6S RNA
Function antagonist of SigA
Interactions involving this RNA in SubtInteract: BsrA
Gene length 190 bp
Immediate neighbours yrvM, aspS
Gene sequence (+200bp)
Genetic context
BsrA context.jpeg








Categories containing this gene/protein

sigma factors and their control, ncRNA

This gene is a member of the following regulons

The gene

Basic information

  • Locus tag:

Phenotypes of a mutant

  • reduced growth rate PubMed
  • delayed outgrowth from stationary phase PubMed

Database entries

  • DBTBS entry: no entry
  • SubtiList entry: no entry
  • Structure:

Additional information

Expression and regulation

  • Regulation: expressed in vegetative cells PubMed
  • Regulatory mechanism:
  • Additional information: transcribed as 201 nt RNA, processed to mature 190 bp 6S RNA PubMed

Biological materials

  • Mutant:
  • Expression vector:
  • lacZ fusion:


Labs working on this gene/RNA

Kouji Nakamura, Tsukuba, University, Japan

Roland Hartmann, Philipps-Universität Marburg, Marburg, Germany, Homepage

Your additional remarks

References

Additional publications: PubMed