Difference between revisions of "Transcription factors modulated by trigger enzymes"
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==Transcription factors controlled by regulatory interactions with [[trigger enzymes|trigger enzyme]]== | ==Transcription factors controlled by regulatory interactions with [[trigger enzymes|trigger enzyme]]== | ||
| − | ** The interaction results in loss of DNA-binding activity of the transcription factor | + | '''** The interaction results in loss of DNA-binding activity of the transcription factor''' |
* [[GltC]]: controlled by [[RocG]] or [[GudB]] (the latter in non-domesticated strains) | * [[GltC]]: controlled by [[RocG]] or [[GudB]] (the latter in non-domesticated strains) | ||
* [[TnrA]]: controlled by [[GlnA]] | * [[TnrA]]: controlled by [[GlnA]] | ||
| − | ** The interaction results in gain of DNA-binding activity of the transcription factor | + | '''** The interaction results in gain of DNA-binding activity of the transcription factor''' |
| − | * [[GlnR]]: | + | * [[GlnR]]: [[GlnA]] acts as co-repressor |
| + | * [[CymR]]: [[CysK]] acts as co-repressor | ||
Revision as of 12:45, 3 November 2009
The activity of these transcription factors is controlled by either modification by or regulatory interaction with trigger enzymes in response to specific signals (usually nutrient availability).
Transcription factors controlled by trigger enzyme-dependent covalent modification
- GlcT: controlled by PtsG
- LicT: controlled by BglP
- SacT: controlled by SacP
- SacY: controlled by SacX
- LevR: controlled by LevE
- LicR: controlled by LicB
- ManR: controlled by ManP
- MtlR: controlled by MtlA
Transcription factors controlled by regulatory interactions with trigger enzyme
** The interaction results in loss of DNA-binding activity of the transcription factor
** The interaction results in gain of DNA-binding activity of the transcription factor
